grant
Synaptic Signaling Mechanisms in Toxoplasma Infected Brains
Organization VIRGINIA POLYTECHNIC INST AND ST UNIVLocation BLACKSBURG, UNITED STATESPosted 15 May 2026Deadline 30 Apr 2031
NIHUS FederalResearch GrantFY2026AIDSAIDS patientsAbscissionAcquired Immune DeficiencyAcquired Immune Deficiency SyndromeAcquired Immunodeficiency SyndromeAdvanced HIVAntibodiesAutoregulationBlood - brain barrier anatomyBlood-Brain BarrierBody TissuesBrainBrain InflammationBrain Nervous SystemCancer PatientCell BodyCell CommunicationCell Communication and SignalingCell InteractionCell SignalingCell-to-Cell InteractionCellsChemoattractantsChemotactic FactorsChemotaxinsChronicClinicalComplementComplement ProteinsDataDiseaseDisorderEncephalitisEncephalonEncephalopathiesEnvironmentEpilepsyEpileptic SeizuresEpilepticsEventExcisionExcitatory SynapseExtirpationFetusFutureGeneralized GrowthGoalsGrowthHemato-Encephalic BarrierHomeostasisHortega cellIL-1IL-1 ReceptorsIL1IL1 ReceptorsImmune responseImmunocompromisedImmunocompromised HostImmunocompromised PatientImmunosuppressed HostIndividualInfectionInflammationInflammatoryInflammatory ResponseInhibitory SynapseInterleukin IInterleukin-1Interleukin-1 ReceptorsIntracellular Communication and SignalingLeadLong-term infectionLymphocyte-Stimulating HormoneMacrophage Cell FactorMediatingMiceMice MammalsMicrogliaModelingMurineMusNerve CellsNerve Impulse TransmissionNerve TransmissionNerve UnitNeural CellNeural TransmissionNeurocyteNeurologicNeurologicalNeuronal TransmissionNeuronsParasite ControlParasitesPatient outcomePatient-Centered OutcomesPatient-Focused OutcomesPatientsPb elementPersonsPhagocytesPhagocytic CellPhysiological HomeostasisPopulationProcessProteinsReceptor ProteinReceptor SignalingRemovalReportingSeizure DisorderSeizuresSevere HIV DiseaseSignal TransductionSignal Transduction SystemsSignalingSourceSurgical RemovalSynapsesSynapticSynaptic TransmissionT Helper FactorT gondiiT gondii infectionT. gondiiT. gondii infectionTestingTissue GrowthTissuesToxoplasmaToxoplasma gondiiToxoplasma gondii InfectionToxoplasmosisWorkacquired immunodeficiency syndrome patientamebocyteaxon signalingaxon-glial signalingaxonal signalingbiological signal transductionbloodbrain barrierchronic infectioncomplement chemotactic factorcomplement systemcomplementationcytokineepilepsiaepileptiformepileptogenicexcitatory neuronexcitotoxicexcitotoxicitygitter cellglia signalingglial signalingglutamate signalingglutamatergic dendrodendritic synapsesglutamatergic signalingheavy metal Pbheavy metal leadhost responsehypoimmunityimmune deficiencyimmune system responseimmunodeficiencyimmunoresponseimmunosuppressed patientlymphocyte activating factormesogliamicroglial cellmicrogliocytenerve signalingneural cell bodyneural signalingneuronalneuronal cell bodyneuronal signalingneurotransmissionnew drug treatmentsnew drugsnew pharmacological therapeuticnew therapeuticsnew therapynext generation therapeuticsnovel drug treatmentsnovel drugsnovel pharmaco-therapeuticnovel pharmacological therapeuticnovel therapeuticsnovel therapyontogenypathogenpatient oriented outcomespatients infected with AIDSpatients with AIDSpatients with acquired immunodeficiency syndromeperivascular glial cellpersistent infectionreceptorrecruitresectionresponsesomasynapsetoxoplasma encephalitistoxoplasmic encephalitis
Description preview
The protozoan parasite Toxoplasma gondii infects approximately one-third of the
world’s population. Most people are asymptomatic because the parasite resides
latently within the brain and other tissues. But AIDS and other
immunosuppressed patients as well as fetuses are in danger of developing
toxoplasmosis when the parasite reactivates and the…
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