grant
Staphylococcus aureus Survival During Nutrient Restriction and Suppression of Host Immunity.
Organization UNIVERSITY OF ILLINOIS AT CHICAGOLocation Chicago, UNITED STATESPosted 15 Jan 2016Deadline 31 Mar 2031
NIHUS FederalResearch GrantFY20261,2-Dithiolane-3-pentanoic acidADP RiboseADP Ribose TransferasesADP ribosylationADP-RibosyltransferaseADPRTsART TransferasesARTasesAbscissionAdenosine 5'-(trihydrogen diphosphate), P'-5-ester with D-riboseAdenosine 5'-DiphosphoriboseAdenosine Diphosphate RiboseAdenosine DiphosphoriboseAminoacetic AcidAnabolismAreaAssayAutoregulationBacteriaBioassayBiologicalBiological AssayBiologyBody TissuesCarrier ProteinsCell BodyCell Communication and SignalingCell FunctionCell PhysiologyCell ProcessCell SignalingCellsCellular FunctionCellular PhysiologyCellular ProcessCommunitiesCysteineDiseaseDisorderDissectionEnvironmentEnzyme GeneEnzymesEukaryotaEukaryoteExcisionExtirpationFirmicutesFoundationsFundingGene ExpressionGeneralized GrowthGenesGlycineGrantGrowthHalf-CystineHomeostasisHospital InfectionsHospital acquired infectionHydroxylasesImmuneImmune responseImmunesImmunityImmunoblottingIn VitroInfectionIntermediary MetabolismIntracellular Communication and SignalingInvestigationL-CysteineLigaseLigase GeneLipoic AcidMechanicsMediatorMetabolicMetabolic ProcessesMetabolismMixed Function OxidasesMixed Function OxygenasesModificationMolecularMonooxygenasesMultienzyme ComplexesNosocomial InfectionsNutrientNutritionalOperonOxidation-ReductionOxidative BurstOxidative StressPathogenesisPathogenicityPathway interactionsPhysiologicPhysiologicalPhysiological HomeostasisPositionPositioning AttributePost-Translational Modification Protein/Amino Acid BiochemistryPost-Translational ModificationsPost-Translational Protein ModificationPost-Translational Protein ProcessingPosttranslational ModificationsPosttranslational Protein ProcessingProtein ModificationProteinsRecoveryRedoxRemovalResearchResistanceResistance to infectionRespiratory BurstRoleS aureusS. aureusSignal TransductionSignal Transduction SystemsSignalingSiteStaph aureusStaphylococcus aureusSubcellular ProcessSurgical RemovalSynthetasesTIL4TLR2TLR2 geneTLR2 receptorTestingThioctic AcidTissue GrowthTissuesToll-Like Receptor 2Toll/Interleukin 1 Receptor-Like 4Toll/Interleukin 1 Receptor-Like 4 GeneToll/Interleukin 1 Receptor-Like Protein 4ToxinTransport Protein GeneTransport ProteinsTransporter ProteinUnited StatesVirulenceWestern BlottingWestern ImmunoblottingWorkalpha-Lipoic Acidbacteria pathogenbacterial pathogenbiologicbiological adaptation to stressbiological signal transductionbiosynthesiscofactorcombatcomparativeenzyme activityenzyme complexhost microbe associationhost microbe relationshiphost responsehost-microbe interactionshost-microbial interactionshost-microorganism interactionsimmune system responseimmunoresponsein vivoin vivo Modelinfection resistanceinstitutional infectionleukocyte oxidative burstmechanicmechanicalnutritiousontogenyoxidationoxidation reduction reactionoxidative damageoxidative injurypathogenpathogenic bacteriapathwaypreventpreventingprotein blottingreaction; crisisresectionresistantsocial rolestemstress responsestress; reactionstressor
Description preview
Project Summary
Staphylococcus aureus is a leading cause of nosocomial infection in the United States and is a predominant
pathogen in communities. S. aureus survives during infection by subverting immune defenses and adapting to
host-imposed nutrient restriction. Yet, we lack a unifying understanding of how these adaptations promote
survival in…
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