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Staphylococcus aureus Survival During Nutrient Restriction and Suppression of Host Immunity.
Organization UNIVERSITY OF ILLINOIS AT CHICAGOLocation Chicago, UNITED STATESPosted 15 Jan 2016Deadline 6 May 2026 โ ๏ธ
NIHUS FederalResearch GrantFY20251,2-Dithiolane-3-pentanoic acidADP ribosylationAffectAminoacetic AcidAnabolismAntibiotic ResistanceAntioxidantsAutoregulationBacteriaBody TissuesBypassCommunitiesCoupledDKFZp547I0610DKFZp564I0682DehydrogenasesDependenceDevelopmentDisparateEnvironmentEnzyme GeneEnzymesGeneralized GrowthGenerationsGlycineGoalsGrantGrowthHomeostasisHospital InfectionsHospital acquired infectionHydroxylasesImmuneImmune Cell ActivationImmune EvasionImmune responseImmunesImmunityImmunosuppressionImmunosuppression EffectImmunosuppressive EffectIncidenceInfectionInfectious Skin DiseasesInnate Immune ResponseInnate ImmunityIntermediary MetabolismKIAA0012KnowledgeLigandsLigaseLigase GeneLinkLipidsLipoic AcidLuciferase ImmunologicLuciferasesMacrophageMacrophage ActivationMechanicsMediatingMediatorMetabolicMetabolic PathwayMetabolic ProcessesMetabolismMiceMice MammalsMixed Function OxidasesMixed Function OxygenasesMonooxygenasesMorbidityMorbidity - disease rateMultienzyme ComplexesMurineMusMฯNADP DehydrogenaseNADP DiaphoraseNADPH DehydrogenaseNADPH DiaphoraseNative ImmunityNatural ImmunityNitrogenNon-Specific ImmunityNonspecific ImmunityNosocomial InfectionsNutrientNutritionalO elementO2 elementOld Yellow EnzymeOxidation-ReductionOxidative StressOxidoreductaseOxidoreductase GeneOxygenPathway interactionsPeptidesPhysiological HomeostasisPrevalenceProcessProductionProteinsPublic HealthPyruvate Dehydrogenase ComplexRedoxReductasesRegulationResistanceResistance to antibioticsResistant to antibioticsRoleS aureusS. aureusS. aureus infectionSilent Mating Type Information Regulator 2-like ProteinsSir2-like ProteinsSirtuinsSiteSkinStaph aureusStaph aureus infectionStaphylococcus aureusStaphylococcus aureus infectionSynthetasesSystemSystemic infectionTLR1TLR1 geneTLR1 proteinTLR1 receptorTPN DiaphoraseTherapeuticThioctic AcidTissue GrowthTissuesToll-Like Receptor 1Toll/Interleukin-1 Receptor-LikeUnited StatesVirulenceVirulentWorkalpha-Lipoic Acidantibiotic drug resistanceantibiotic resistantbacteria metabolismbacterial metabolismbiological adaptation to stressbiosynthesisblood infectionbloodstream infectioncell killingcofactorcutaneous infectioncytokinedevelopmentalenzyme complexfitnesshost responseimmune activationimmune evasiveimmune suppressionimmune suppressive activityimmune suppressive functionimmune system responseimmunoresponseimmunosuppressive activityimmunosuppressive functionimmunosuppressive responseimprovedin vivoinfected skininfected with S. aureusinfected with Staph aureusinfected with Staphylococcus aureusinfection in the bloodinfection of the bloodinstitutional infectionintraperitonealmechanicmechanicalmortalitymutantnew drug targetnew drug treatmentsnew druggable targetnew drugsnew pharmacological therapeuticnew pharmacotherapy targetnew therapeutic targetnew therapeuticsnew therapynew therapy targetnew vaccinesnext generation therapeuticsnext generation vaccinesnovelnovel drug targetnovel drug treatmentsnovel druggable targetnovel drugsnovel pharmaco-therapeuticnovel pharmacological therapeuticnovel pharmacotherapy targetnovel therapeutic targetnovel therapeuticsnovel therapynovel therapy targetnovel vaccinesnutritiousontogenyoxidation reduction reactionpathogenpathwaypreventpreventingrational designreaction; crisisrecruitrelease factorresistantresponsersc786skin infectionsocial rolestress responsestress; reactiontreatment strategy
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Project Summary
Staphylococcus aureus (Sa) is a leading cause of nosocomial infection in the United States and is a predominant
pathogen in communities. Treatment of Sa infections is complicated by the prevalence of antibiotic resistant and
highly virulent clones, making new therapeutic alternatives a necessity. Sa survives during infection byโฆ
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