grant
Nutritional Control of Nematode Development
NIHUS FederalResearch GrantFY20251-Phosphatidylinositol 3-Kinase21+ years oldAcuteAdultAdult HumanAffectAgingAnatomic SitesAnatomic structuresAnatomyAnimal ModelAnimal Models and Related StudiesAnimalsAutoregulationBiochemistryBiological ChemistryBody TissuesC elegansC. elegansC.elegansCaenorhabditis elegansCancer CauseCancer EtiologyCancer ModelCancerModelCell Communication and SignalingCell SignalingDNA Polymerase IIDNA Polymerase epsilonDNA-Dependent DNA Polymerase IIDevelopmentDiabetes MellitusDietDiseaseDisorderErinaceidaeExtracellular Signal-Regulated Kinase GeneFoodGene ExpressionGene TranscriptionGeneralized GrowthGenesGenetic TranscriptionGerm LinesGoalsGrowthHealthHedgehogsHomeostasisHumanHumulin RInstructionInsulinIntermediary MetabolismIntracellular Communication and SignalingKnowledgeLINLifeLipid TraffickingLipidsLipoproteinsLobular Intraepithelial NeoplasiaLobular NeoplasiaMAP Kinase GeneMAPKMMAC1MMAC1 proteinMediatingMetabolic ProcessesMetabolismMitogen-Activated Protein Kinase GeneModelingModern ManMolecularMutated in Multiple Advanced Cancers 1NematodaNematodesNon-Polyadenylated RNANovolin RNutrientNutrient availabilityNutritionalOocytesOvocytesPHTS genePHTS proteinPI-3 KinasePI3-KinasePI3CGPI3KGammaPI3kPIK3PIK3CGPIK3CG genePTENPTEN genePTEN proteinPTEN1PathologicPathway interactionsPhosphatase and Tensin HomologPhosphatase and Tensin Homolog Deleted on Chromosome 10Phosphatidylinositol 3-KinasePhosphatidylinositol-3-OH KinasePhosphoinositide 3-HydroxykinasePhosphoprotein PhosphatasePhosphoprotein Phosphatase-2CPhosphoprotein PhosphohydrolasePhysiological HomeostasisPol IIPreventative strategyPrevention strategyPreventive strategyProtein Phosphatase CProtein Phosphatase GeneProtein Phosphatase-1Protein Phosphatase-2AProtein phosphatasePtdIns 3-KinasePublic HealthRNARNA ExpressionRNA Gene ProductsRegular InsulinRegulationResearchResistanceResolutionRibonucleic AcidRoleSignal PathwaySignal TransductionSignal Transduction SystemsSignalingStarvationTherapeutic InterventionTissue GrowthTissuesTranscriptionTumor Suppressor ProteinsType I Phosphatidylinositol KinaseType III Phosphoinositide 3-KinaseVitellogeninsWorkadulthoodautomated image analysisbiological signal transductiondevelopmentaldiabetesdietsfunctional genomicsgenetic analysishatchinginnovateinnovationinnovativeintervention therapylipid transportmodel of animalmutated in multiple advanced cancers 1 proteinnovelnutritiousontogenypathwayphosphatase and tensin homologue on chromosome tenresistance mechanismresistantresistant mechanismresolutionsresponseroundwormsocial roletraffickingtumortumor suppressor
Description preview
SUMMARY
Nutrient-responsive pathways govern gene expression and metabolism to adapt to starvation, and their
dysregulation causes cancer, diabetes, and aging. The Baugh lab uses C. elegans to investigate how animals
maintain developmental homeostasis despite fluctuations in nutrient availability. C. elegans is a premier animal
model to study…
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