grant
Modeling extensive cellular variability in glycolytic rates using multiplexed live-cell data
Organization UNIVERSITY OF CALIFORNIA AT DAVISLocation DAVIS, UNITED STATESPosted 1 Apr 2026Deadline 28 Feb 2031
NIHUS FederalResearch GrantFY20261-Phosphatidylinositol 3-KinaseAKTAffectAgingAkt proteinApoptosisApoptosis PathwayAssayAutoregulationBehaviorBioassayBiological AssayBiosensorBody TissuesCancersCell BodyCell Communication and SignalingCell Culture TechniquesCell DeathCell DifferentiationCell Differentiation processCell FunctionCell Growth in NumberCell LineCell MultiplicationCell PhysiologyCell ProcessCell ProliferationCell SignalingCell modelCellLineCellsCellular FunctionCellular Immune FunctionCellular Metabolic ProcessCellular PhysiologyCellular ProcessCellular ProliferationCellular RegulationCellular modelCessation of lifeComputer ModelsComputerized ModelsD-GlucoseDataDeathDextroseDiabetes MellitusDiseaseDisorderEndocrineEpithelial CellsEpitheliumFK506 Binding Protein 12-Rapamycin Associated Protein 1FKBP12 Rapamycin Complex Associated Protein 1FRAP1FRAP1 geneFRAP2FructoseGene ExpressionGeneticGlucoseGlycolysisGlycolysis PathwayHealthHepatic CellsHepatic Parenchymal CellHepatocyteHeterogeneityHistoryHomeostasisHumanHumulin RImmuneImmunesIndividualInsulinIntermediary MetabolismIntracellular Communication and SignalingInvestigationKinasesLactic acidLevuloseLifeLinkLiverLiver CellsMalignant CellMalignant NeoplasmsMalignant TumorMath ModelsMeasurementMeasuresMechanistic Target of RapamycinMetabolicMetabolic DiseasesMetabolic DisorderMetabolic PathwayMetabolic ProcessesMetabolic dysfunctionMetabolismMethodsMicroscopicModelingModern ManMolecularMuscleMuscle CellsMuscle FibersMuscle TissueMyocytesMyotubesNADHNormal CellNovolin RPI-3 KinasePI3-KinasePI3CGPI3KGammaPI3kPIK3PIK3CGPIK3CG genePathway interactionsPhosphatidylinositol 3-KinasePhosphatidylinositol-3-OH KinasePhosphoinositide 3-HydroxykinasePhosphotransferase GenePhosphotransferasesPhysiological HomeostasisPlayPopulationPopulation DistributionsPositionPositioning AttributeProcessProductionProgrammed Cell DeathProtein BiosynthesisProtein Kinase BProto-Oncogene Proteins c-aktPtdIns 3-KinasePyruvateRAC-PK proteinRAFT1Recording of previous eventsRegular InsulinRegulationRhabdomyocyteRibosomal Peptide BiosynthesisRibosomal Protein BiosynthesisRibosomal Protein SynthesisRoleSignal PathwaySignal TransductionSignal Transduction SystemsSignalingSignaling Factor Proto-OncogeneSignaling Pathway GeneSignaling ProteinSkeletal FiberSkeletal Muscle CellSkeletal Muscle FiberSkeletal MyocytesStatistical MethodsStrains Cell LinesStructureSubcellular ProcessSystemTestingThesaurismosisTimeTissuesTransphosphorylasesType I Phosphatidylinositol KinaseType III Phosphoinositide 3-KinaseVariantVariationactivity markerbiological sensorbiological signal transductionc-akt proteincancer cellcell culturecell culturescell growth regulationcell metabolismcell typecellular differentiationcellular metabaolismcomputational modelingcomputational modelscomputer based modelscomputer based predictioncomputerized modelingcultured cell linediabetesdrug efficacyglucose disposalglucose uptakehepatic body systemhepatic organ systemhistorieshuman tissueimmune functionin vivoinhibitorinsulin stimulated glucose disposalmTORmalignancymammalian target of rapamycinmathematic modelmathematical modelmathematical modelingmetabolism disordermolecular pathologymuscularnecrocytosisneoplasm/cancerpathwaypharmacologicpredictive modelingprotein synthesisproto-oncogene protein RACproto-oncogene protein aktrac protein kinaserelated to A and C-proteinsensorsocial rolestatistic methodssugartool
Description preview
Summary
Glycolysis, the fundamental metabolic process in which glucose is converted to lactate, lies at the
core of diabetes and metabolic dysfunction, as well as cancer and immune function. Regulation
of glycolytic rate by insulin and other endocrine signals is essential for bodily metabolic
homeostasis and also plays important cell-intrinsic…
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