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Investigating the role of intestinal-derived oxytocin in social behavior and gut inflammation.
Organization BAYLOR COLLEGE OF MEDICINELocation HOUSTON, UNITED STATESPosted 1 Apr 2024Deadline 31 Mar 2026 ⚠️
NIHUS FederalResearch GrantFY20250-11 years old21+ years oldASDAdultAdult HumanAffectAnimal ModelAnimal Models and Related StudiesAnimalsAnti-InflammatoriesAnti-Inflammatory AgentsAnti-inflammatoryAnxietyAutismAutistic DisorderBehaviorBehavioralBody TissuesBody Weight decreasedBrainBrain Nervous SystemCell Communication and SignalingCell Growth in NumberCell MultiplicationCell ProliferationCell SignalingCellular ProliferationChildChild YouthChildren (0-21)ClinicalColitisCommunicationDataDefectDepakoteDepakote ERDiseaseDisorderDivalproexDrugsDysfunctionEarly Infantile AutismEncephalonEndocrine Gland SecretionEpithelial CellsEpitheliumExposure toFamily suidaeFlareFunctional disorderGut EpitheliumHormonesHumanHypothalamic structureHypothalamusImmune systemIncidenceIndividualInfantile AutismInflammationInflammatory Bowel DiseasesInflammatory Bowel DisorderIntestinalIntestinesIntracellular Communication and SignalingInvestigationKO miceKanner's SyndromeKnock-outKnock-out MiceKnockoutKnockout MiceL reuteriL. reuteriLactobacillus reuteriLearningLinkLocationMediatingMedicationMiceMice MammalsMicrobeModern ManMsecMurineMusNerveNull MouseOcytocinOralOxytocinOxytocin ReceptorPathogenicityPathologyPatientsPermeabilityPharmaceutical PreparationsPhenotypePhysiopathologyPigsRecombinant OxytocinResearchRoleSeveritiesSignal TransductionSignal Transduction SystemsSignalingSocial BehaviorSocial InteractionSodium Dextran SulfateStressSuidaeSwineTestingTherapeuticTherapeutic HormoneTissuesTransmissionValproic AcidWeight LossWeight Reductionadulthoodassociated symptomautism modelautism spectral disorderautism spectrum disorderautistic individualsautistic peopleautistic spectrum disorderbiological signal transductionbody weight lossbowelbowel inflammationco-morbid symptomco-occuring symptomcolitis mouse modelcolitis murine modelcomorbid symptomconcurrent symptomcooccuring symptomdosagedrug/agentexposed in uterofetal exposuregastrointestinalgastrointestinal epitheliumgut inflammationgut microbesgut microbial speciesgut to brain axisgut-brain axisgut-brain communicationgut-brain interactionsgut-brain relationshipgut-brain signalinghigh rewardhigh riskhypothalamicin utero exposureindividuals on the autism spectrumindividuals on the spectrumindividuals with ASDindividuals with autismindividuals with autism spectrum disorderinflamed bowelinflamed gutinflamed intestineinflammatory disease of the intestineinflammatory disorder of the intestineintestinal autoinflammationintestinal epitheliumintestinal inflammationintestinal microbesintra-uterine environmental exposureintrauterine environmental exposurekidsmicrobialmicrobiomemillisecondmodel of animalmodel of autism spectrum disordermouse colitismouse modelmurine colitismurine modelnovelpathophysiologypeople on the autism spectrumpeople with ASDpeople with autismpeople with autism spectrum disorderporcineprenatal exposureprenatally exposedremediationrepetitive behaviorsocialsocial rolesocial stresssocially stressedsociobehaviorsociobehavioralsuccesssuidsymptom associationsymptom comorbiditytransmission processwt-lossyoungster
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Description preview
ABSTRACT
The gut-brain axis has been recognized as a highway sending beneficial and pathogenic signals in the gut in
milliseconds to the brain and the body. This gut-brain connection may explain the overlap in seemingly
different diseases, namely the overlap between autism spectrum disorder (ASD) and gastrointestinal
dysfunctions including…
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