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Host-pathogen interactions during GBS diabetic wound infections
Organization UNIVERSITY OF COLORADO DENVERLocation Aurora, UNITED STATESPosted 1 Jun 2024Deadline 15 Aug 2025 ⚠️
NIHUS FederalResearch GrantFY20252-oxo-propanal21+ years old27E10 AntigenABC Transport ProteinABC Transporter ProteinABC TransportersATP-Binding Cassette TransportersAcetylformaldehydeAddressAdultAdult HumanAdvanced Glycation End ProductsAdvanced Glycosylation End ProductsAssayAutoregulationBacteriaBacterial InfectionsBindingBioassayBiological AssayBiophysicsBlood NeutrophilBlood PlasmaBlood Polymorphonuclear NeutrophilBlood SerumCAGBCGCBCalcium-Binding Myeloid Protein P8,14CalgranulinCalorimetryCalprotectinCause of DeathCellular Immune FunctionComplications of Diabetes MellitusCoupledD-GlucoseDataData SetDextroseDiabetes ComplicationsDiabetes MellitusDiabetes-Related ComplicationsDiabetic ComplicationsDiabetic mouseDiabetic woundDiffusionDivalent CationsDrug Metabolic DetoxicationDrug Metabolic DetoxificationE coliE. coliEnzyme GeneEnzymesEscherichia coliGene TranscriptionGeneralized GrowthGenetic TranscriptionGlucoseGlycolysisGlyoxalase IGrowthHL-60 CellsHL60 CellsHealthHemi-MyeloperoxidaseHomeostasisHumanImmune responseIn VitroIndividualInfectionInfectious Skin DiseasesInflammationInflammatoryIntermediary MetabolismInvadedL1 AntigenLIAGLactoyl Glutathione LyaseLactoylglutathione LyaseLeukocyte L1 Antigen ComplexLeukocyte L1 ProteinMAC387MRP14ManganeseMarrow NeutrophilMeasuresMediatingMetabolic Drug DetoxicationsMetabolic ProcessesMetabolismMetabolism of Toxic AgentsMetalsMethylglyoxalMethylglyoxalaseMiceMice MammalsMigratory Inhibitory Factor-Related Protein MRPMinimum Inhibitory Concentration measurementMinimum Inhibitory ConcentrationsMn elementModelingModern ManMolecular InteractionMonitorMurineMusMyelomonocytic Antigen L1MyeloperoxidaseNeonatal meningitisNeuropathyNeutrophilic GranulocyteNeutrophilic LeukocyteNi elementNickelNutrientNutritional ImmunityOrganismOutcomePathogenesisPathway interactionsPeroxidasesPersonsPhysiological HomeostasisPlasmaPlasma SerumPolymorphonuclear CellPolymorphonuclear LeukocytesPolymorphonuclear NeutrophilsPopulationPredispositionProcessProductionProteinsPyruvaldehydePyruvic AldehydeRNA ExpressionRNA SeqRNA sequencingRNAseqReticuloendothelial System, Serum, PlasmaRoleS agalactiaeS. agalactiaeS100A9S100A9 geneSTZSerumSkinSoft Tissue InfectionsSpecificityStreptococcal InfectionsStreptococcus Group BStreptococcus agalactiaeStreptococcus infectionStreptozocinStreptozotocinStressStructureSusceptibilitySystemTherapeuticTissue GrowthTitrationsToxinTranscriptionVascular DiseasesVascular DisorderWhole BloodWorkWound InfectionWound modelsZanosarZincZn elementadulthoodadvanced glycation endproductadvanced glycosylation endproductbacteria infectionbacterial diseasebiophysical foundationbiophysical principlesbiophysical sciencesblood vessel disorderchronic skin woundchronic woundcofactorcutaneous infectiondetoxificationdiabetesdiabetes mouse modeldiabetes ulcerdiabeticdiabetic skin wounddiabetic ulcerdiffuseddiffusesdiffusingdiffusionsfemale genital tractfemale reproductive tractgastrointestinalglyoxalasehealinghost responseimmune functionimmune system responseimmunoresponsein vivo Modelinfected skininfected woundliving systemmetal chelatorneonateneuropathicneutrophilnovelontogenyoptical emission spectroscopypathobiontpathogenpathwayresponseskin infectionsocial rolesubcutaneoussubdermaltissue woundtranscriptome sequencingtranscriptomic sequencingvascular dysfunctionvasculopathywomen's genital tractwomen's reproductive tractwoundwound environmentwound healing modelswoundingwounds
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Description preview
PROJECT SUMMARY
Group B Streptococcus (GBS), is a Gram-positive pathobiont that commonly colonizes the gastrointestinal and
lower female reproductive tracts but can cause adverse health outcomes in neonates and vulnerable adult
populations. GBS has also been increasingly associated with diabetic wound infections and is the third leading
cause of…
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