grant
Dysfunctional mechanisms of the mitochondrial redox-sensitive signaling response to exercise in aged skeletal muscle
Organization UNIVERSITY OF WASHINGTONLocation SEATTLE, UNITED STATESPosted 1 Sept 2025Deadline 31 May 2030
NIHUS FederalResearch GrantFY20253-D3-D modeling3-Dimensional3D3D modelingAcuteAffectAgeAgingAntioxidantsAssayBioassayBioenergeticsBiological AssayBiologyBiology of AgingBody TissuesCareer Development AwardsCareer Development Awards and ProgramsCareer Development Programs K-SeriesCell Communication and SignalingCell LineCell SignalingCellLineChronicCirculatory CollapseCitric Acid CycleCollaborationsCompensationComplementComplement ProteinsComplexDataDefectDevelopmentDevelopment PlansDoctor of PhilosophyDysfunctionEconomic BurdenEmbryonic Muscle CellsEnvironmentEnzyme InteractionExerciseFatsFatty acid glycerol estersFoundationsFunctional disorderFundingFutureGeneral RadiologyGoalsGrantHealth Care CostsHealth CostsHealth ExpendituresHospital AdmissionHospitalizationHumanImpairmentIn VitroIndividualIntermediary MetabolismIntracellular Communication and SignalingK-AwardsK-Series Research Career ProgramsKrebs CycleLabelLaboratory ResearchLifeLinkLipidsMass Photometry/Spectrum AnalysisMass SpectrometryMass SpectroscopyMass SpectrumMass Spectrum AnalysesMass Spectrum AnalysisMediatingMentorsMentorshipMetabolicMetabolic PathwayMetabolic ProcessesMetabolismMitochondriaMitochondrial ProteinsModern ManModificationMuscleMuscle AtrophyMuscle Cell ContractionMuscle ContractionMuscle MitochondriaMuscle TissueMuscle WeaknessMuscle functionMuscular AtrophyMuscular ContractionMuscular WeaknessMyoblastsMyoneural JunctionNeuromuscular JunctionNoiseNutrientOrganellesOxidantsOxidation-ReductionOxidative StressOxidizing AgentsPathologicPathologyPeptidesPh.D.PhDPhenotypePhysiopathologyPlayPrecursor Muscle CellsProductionProteinsRadiologyRadiology SpecialtyReceptor ProteinRedoxRelaxationResearchResearch Career ProgramRoleSarcosomesScheduleShockSignal TransductionSignal Transduction SystemsSignalingSkeletal MuscleStrains Cell LinesStressSystemTCA cycleTRX geneTRX proteinTRX1TXN geneTechniquesTestingTherapeuticThioredoxinTissue EngineeringTissuesTrainingTricarboxylic Acid CycleUniversitiesVoluntary MuscleWashingtonWorkWritingage associatedage associated alterationsage associated changesage associated effectsage correlatedage correlated alterationsage correlated changesage dependentage dependent alterationsage dependent changesage effectage induced alterationsage induced changesage linkedage relatedage related alterationsage related changesage related effectsage specificage specific alterationsage specific changesagedaged animalaged animalsaged groupaged groupsaged individualaged individualsaged muscleaged peopleaged personaged personsaged populationaged populationsagesaging associatedaging associated alterationsaging associated changesaging correlated alterationsaging correlated changesaging dependent alterationsaging dependent changesaging effectaging induced alterationsaging induced changesaging of muscleaging populationaging relatedaging related alterationsaging related changesaging specific alterationsaging specific changesalterations with ageanimal old agebioengineered tissuebiological signal transductionblood glucose regulationcareer developmentchanges with agecirculatory shockcomplementationconferenceconventioncostcrosslinkcultured cell linedesigndesigningdevelopmentalelderly animalelectron acceptorengineered tissueexercise trainingexperiencefatty acid oxidationfull professorgene manipulationgenetic manipulationgenetically manipulategenetically perturbglucose controlglucose homeostasisglucose metabolismglucose regulationglutathione peroxidasehealth care expenditurehiPSChuman iPShuman iPSChuman induced pluripotent cellhuman induced pluripotent stem cellshuman inducible pluripotent stem cellshuman inducible stem cellsiPSiPSCiPSCsimpact of ageimprovedin vivoinduced human pluripotent stem cellsinduced pluripotent cellinduced pluripotent stem cellinducible pluripotent cellinducible pluripotent stem cellinfluence of ageinsightlarge data setslarge datasetslecturesmedical expendituremeetingmeetingsmembermetabolic phenotypemetabolism measurementmetabolomicsmetabonomicsmetabotypemitochondrialmitochondrial metabolismmouse modelmurine modelmuscle agingmuscle breakdownmuscle bulkmuscle degradationmuscle deteriorationmuscle engineeringmuscle formmuscle lossmuscle massmuscle metabolismmuscle wastingmuscularnovelold animalsoxidationoxidation reduction reactionoxidative damageoxidative injurypathophysiologyperoxiredoxinpharmacologicpopulation agingpreventpreventingprogramsprotein expressionreceptorresistance exerciseresistance trainingresponsesarcopeniasarcopenicsenior facultyshocksskeletal muscle atrophyskeletal muscle breakdownskeletal muscle lossskeletal muscle protein lossskeletal muscle wastingsocial rolesummitsymposiasymposiumtargeted drug therapytargeted drug treatmentstargeted therapeutictargeted therapeutic agentstargeted therapytargeted treatmentthree dimensionalthree-dimensional modelingtool
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Abstract: Dysfunctional mechanisms of the mitochondrial redox-sensitive signaling response to exercise in
aged skeletal muscle.
Individual: This career development award application details training and scientific research for Matthew D.
Campbell, Ph.D. a skeletal muscle and mitochondrial biologist in the Radiology department at the University of…
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