grant
Contaminant Metal Content in Wildfire Smoke and Neuroinflammation
Organization UNIVERSITY OF NEW MEXICO HEALTH SCIS CTRLocation ALBUQUERQUE, UNITED STATESPosted 1 Aug 2016Deadline 31 Jan 2028
NIHUS FederalResearch GrantFY2025AccelerationAcclimatizationAcuteAddressAdhesion MoleculeAgingAreaArizonaArsenicAstrocytesAstrocytusAstrogliaAutomobile DrivingBiomassBlood NeutrophilBlood Polymorphonuclear NeutrophilBlood leukocyteBrainBrain Nervous SystemBrain VascularCCL2CCL2 geneCD106CD106 AntigensCD31CD54 AntigensCadmiumCaliforniaCd elementCell Adhesion Molecule GeneCell Adhesion MoleculesCell BodyCell surfaceCellsChemicalsChemokine, CC Motif, Ligand 2ChromiumCirculationCommunitiesConsumptionCr elementDataDevelopmentDietElderlyEncephalonEndothelial CellsEndotheliumEventExhibitsExposure toFire - disastersFiresFlow CytofluorometriesFlow CytofluorometryFlow CytometryFlow MicrofluorimetryFlow MicrofluorometryFoundationsFrequenciesHortega cellHouseholdICAM-1INCAM-110ImmuneImmune infiltratesImmunesInducible Cell Adhesion Molecule 110InflammationInflammatoryInhalationInhalation ExposureInhalingInjuryIntercellular adhesion molecule 1KnowledgeLaboratoriesLearningLeukocytesLeukocytes Reticuloendothelial SystemLung InflammationMCAFMCP-1MCP1Marrow NeutrophilMarrow leukocyteMetal exposureMetalsMiceMice MammalsMicrogliaMiningModelingMolecularMonocyte Chemoattractant Protein-1Monocyte Chemotactic Protein-1Monocyte Chemotactic and Activating FactorMonocyte Chemotactic and Activating ProteinMonocyte Chemotactive and Activating FactorMonocyte Secretory Protein JEMurineMusNatureNavahoNavajoNerve DegenerationNeuron DegenerationNeutrophilic GranulocyteNeutrophilic LeukocyteNew MexicoNi elementNickelNuclearOutcomePECAM1PECAM1 geneParticulate MatterPathway interactionsPeripheralPersonsPhenotypePneumonitisPolymorphonuclear CellPolymorphonuclear LeukocytesPolymorphonuclear NeutrophilsPuebloPueblo RacePulmonary InflammationResolutionRiskRoleSCYA2SamplingSaturated Fatty AcidsSeveritiesSiteSmall Inducible Cytokine A2SmokeSourceStimulusSystemTestingTherapeuticTherapeutic InterventionTimeToxic effectToxicitiesU elementUraniumUrineUtahV elementVCAMVCAM-1VanadiumVascular Cell Adhesion MoleculeVascular Cell Adhesion Molecule-1White Blood CellsWhite CellWildfireWoodWood materialWorkadvanced ageaged groupaged groupsaged individualaged individualsaged peopleaged personaged personsaged populationaged populationsaging populationanthropogenesisanthropogenicarsenicsastrocytic gliabiomass fuelcell adhesion proteincerebral vascularcerebro-vascularcerebrovascularclimate changeclimatic changesdevelopmentaldietarydietsdrivingendothelial dysfunctionenvironmental stressesenvironmental stressorexposure to metalfirefirst responderflow cytophotometrygeriatricgitter cellglobal climate changeimmune cell infiltrateinjuriesintervention therapylipid mediatormesogliamicroglial cellmicrogliocyteneural degenerationneural inflammationneuro-vascularneuro-vascular unitneurodegenerationneurodegenerativeneuroinflammationneuroinflammatoryneurological degenerationneuronal degenerationneurovascularneurovascular unitneutrophilpathwayperivascular glial cellpopulation agingpulmonaryrecruitresolutionsresponsesenior citizensmoke inhalationsocial roletoxic metaltrendwhite blood cellwhite blood corpusclewild firewildland firewood smokewoodsmokeyounger age
Description preview
SUMMARY
Wildland fire disasters raged throughout the western US in recent years, continuing a trend for increasing
frequency and severity of such events driven by climate change. Smoke arising from these events covered
much of the continent, exposing over 100 million people. Wildland fires in the New Mexico region can cross
over regions of legacy…
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