grant
SPECIFIC REGULATION OF CARDIAC ATRIAL CONTRACTILITY
Organization LOYOLA UNIVERSITY CHICAGOLocation MAYWOOD, UNITED STATESPosted 10 Sept 2025Deadline 30 Jun 2030
NIHUS FederalResearch GrantFY2025Actin-Activated ATPaseActin-S1 ATPaseActomyosin Subfragment 1 ATPaseAffectAffinityAmino AcidsArrhythmiaAsymmetric Septal HypertrophyAtrialAtrial FibrillationAuricular FibrillationBindingBinding ProteinsBinding SitesC proteinC-terminalCardiacCardiac ArrhythmiaCardiac AtriumCardiac DiseasesCardiac DisordersCardiac Muscle CellsCardiac Muscle MyosinsCardiac MyocytesCardiac MyosinsCardiocyteCardiomyopathiesCell BodyCellsCombining SiteCongestive CardiomyopathyDNA mutationDataDevelopmentDilated CardiomyopathyDiseaseDisorderDysfunctionEquilibriumExpression SignatureFunctional disorderGene Expression ProfileGenerationsGenesGeneticGenetic ChangeGenetic defectGenetic mutationGoalsHeartHeart ArrhythmiasHeart AtriumHeart DiseasesHeart Muscle CellsHeart failureHeart myocyteHeavy Meromyosin Subfragment-1Hereditary ventricular hypertrophyHumanHypertrophic CardiomyopathyHypertrophic Obstructive CardiomyopathyIdiopathic Hypertrophic Subvalvular StenosisIdiopathic hypertrophic subaortic stenosisImpairmentIn VitroKineticsKnock-outKnockoutLeft VentriclesLeft Ventricular FunctionLeft ventricular structureLifeLigand Binding ProteinLigand Binding Protein GeneLinkMeasuresMediatingMiceMice MammalsMissense MutationModelingModern ManMolecular InteractionMorbidityMorbidity - disease rateMurineMusMuscle DiseaseMuscle DisordersMuscular DiseasesMutationMyBP-C proteinMyocardial DiseasesMyocardial DisorderMyocardial depressionMyocardial dysfunctionMyocardiopathiesMyopathic ConditionsMyopathic Diseases and SyndromesMyopathic disease or syndromeMyopathyMyosin ATPaseMyosin Adenosine TriphosphataseMyosin AdenosinetriphosphataseMyosin S-1Myosin Subfragment-1MyosinsN-terminalNH2-terminalNonsense MutationPaste substancePastesPathogenicityPhasePhosphorylationPhysiologicPhysiologicalPhysiopathologyPredispositionPrintingPropertyProtein BindingProtein PhosphorylationProteinsPublishingReactive SiteRegulationRelaxationResearchRoleSarcomeresSusceptibilitySystemTechniquesThick FilamentVariantVariationVentricular DysfunctionVentricular FunctionWorkWritingaminoacidatriumbalancebalance functionbound proteincardiac dysfunctioncardiac failurecardiac functioncardiac myocytes differentiated from induced pluripotent stem cellcardiac tissue engineeringcardiomyocytecartilage link proteincitrate carriercitrate periplasmic carrier proteincitrate transportercitrate-binding transport proteindevelopmentalengineered heart tissueexperimentexperimental researchexperimental studyexperimentsfunction of the heartgene expression patterngene expression signaturegenome mutationheart disorderheart dysfunctionheart functionhiPSChuman iPShuman iPSChuman induced pluripotent cellhuman induced pluripotent stem cellshuman inducible pluripotent stem cellshuman inducible stem cellshypertrophic myocardiopathyiPS cell derived cardiomyocytesiPSC derived cardiomyocytesinduced human pluripotent stem cellsinduced pluripotent stem cell derived cardiac myocytesinduced pluripotent stem cell derived cardiomyocytesinducible pluripotent stem cell derived cardiac myocytesinducible pluripotent stem cells derived cardiomyocyteslink proteinmissense single nucleotide polymorphismmissense single nucleotide variantmissense variantmuscular disordermyocardium diseasemyocardium disordermyosin-binding protein Cnon-sense mutationoverexpressoverexpressionpathophysiologysocial rolestoichiometrytranscriptional profiletranscriptional signaturetricarboxylate carriertricarboxylate transportertricarboxylate-binding C proteinvirtual
Description preview
PROJECT SUMMARY
Cardiomyopathies are a group of diseases, commonly with genetic causes, that impair cardiac function and
can lead to heart failure. The focus of cardiomyopathy research has been left ventricular function, as this
chamber is critical for supporting life, but the whole heart is typically affected by these diseases. The cardiac
atria…
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