grant
Role of microglial phagocytosis in prion diseases
Organization UNIVERSITY OF MARYLAND BALTIMORELocation BALTIMORE, UNITED STATESPosted 30 Sept 2003Deadline 28 Feb 2029
NIHUS FederalResearch GrantFY2026AD dementiaAddressAlzheimer Type DementiaAlzheimer disease dementiaAlzheimer sclerosisAlzheimer syndromeAlzheimer'sAlzheimer's DiseaseAlzheimers DementiaAnimalsApoptoticAwardBiological MarkersCD11bCJDCR3CR3 ReceptorCR3ACell BodyCell Communication and SignalingCell SignalingCellsClinicalComplement 3 ReceptorCreutzfeldt-Jacob DiseaseCreutzfeldt-JakobCreutzfeldt-Jakob DiseaseCreutzfeldt-Jakob SyndromeD-GalactoseDataDegenerative Neurologic DisordersDiseaseDisease ProgressionDisorderEatingEffectivenessEventFDA approvedFlow CytofluorometriesFlow CytofluorometryFlow CytometryFlow MicrofluorimetryFlow MicrofluorometryFood IntakeGalactopyranoseGalactopyranosideGalactoseGliaGlial CellsGoalsHortega cellHumanITGAMITGAM geneIntegrin alpha-M beta-2Integrin alphaMbeta2Intracellular Communication and SignalingJakob-Creutzfeldt DiseaseKnowledgeKolliker's reticulumMAC1AMO1AMac 1Mac-1 Adhesive ReceptorMac-1 AntigenMac-1 ReceptorMacrophage-1 AntigenMediatingMiceMice MammalsMicrogliaMo1 Antigen ReceptorMo1 Glycoprotein ReceptorModern ManMurineMusMyeloid CellsNerve CellsNerve DegenerationNerve UnitNervous System Degenerative DiseasesNeural CellNeural Degenerative DiseasesNeural degenerative DisordersNeurocyteNeurodegenerative DiseasesNeurodegenerative DisordersNeurogliaNeuroglial CellsNeurologic Degenerative ConditionsNeuron DegenerationNeuronsNon-neuronal cellNonneuronal cellOnset of illnessParalysis AgitansParkinsonParkinson DiseasePathogenesisPathway interactionsPhagocytesPhagocytic CellPhagocytosisPhenotypePopulationPrPScPrPSc ProteinsPrimary ParkinsonismPrimary Senile Degenerative DementiaPrion DiseasesPrion Protein DiseasesPrion-Induced DisorderPrionsPurine ReceptorsPurinergic ReceptorsPurinoceptorReceptor ProteinRoleScrapie AgentScrapie PrPSignal TransductionSignal Transduction SystemsSignalingSolidSubacute Spongiform EncephalopathySynaptosomesTerminally IllTestingTherapeuticTherapeutic InterventionTransmissible DementiasTransmissible Spongiform EncephalopathiesWorkamebocytebio-markersbiologic markerbiological signal transductionbiomarkerconditional knock-outconditional knockoutdegenerative diseases of motor and sensory neuronsdegenerative neurological diseasesdesigndesigningdisease onsetdisease-associated PrPdisorder onseteffectiveness testingflow cytophotometrygitter cellinhibitorintervention therapymesogliamicroglial cellmicrogliocytemouse modelmurine modelnerve cementneural degenerationneurodegenerationneurodegenerativeneurodegenerative illnessneurological degenerationneuronalneuronal degenerationpathwayperivascular glial cellprimary degenerative dementiaprion disorderreceptorscreeningscreeningssenile dementia of the Alzheimer typesocial rolespongiform degenerationspongiform encephalopathysynaptoneurosometimelineuptakeαMβ2
Description preview
Prion diseases or Transmissible Spongiform Encephalopathies are transmissible neurodegenerative diseases
in humans and animals that have no treatment and are 100% lethal. The transformation of glia into reactive
states is recognized as one of the major hallmarks of neurodegenerative diseases including prion, Alzheimer’s,
and Parkinson’s diseases.…
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