grant
Multimodal assessment of neural, neuroendocrine, and immune cue responses in binge drinkers in the laboratory and real world.
Organization AUBURN UNIVERSITY AT AUBURNLocation Auburn, UNITED STATESPosted 20 Sept 2025Deadline 31 Aug 2030
NIHUS FederalResearch GrantFY2025(TNF)-α21+ years oldACTHActive Follow-upAdoptionAdrenocorticotropic HormoneAdrenocorticotropinAdultAdult HumanAeroseb-HCAgeAlcohol Chemical ClassAlcohol DrinkingAlcohol consumptionAlcoholsAmericanB cell differentiation factorB cell stimulating factor 2B-Cell Differentiation FactorB-Cell Differentiation Factor-2B-Cell Stimulatory Factor-2BCDFBSF-2BSF2Beta Proprotein Interleukin 1Biological MarkersBlood SampleBlood specimenCachectinCetacortConsumptionCort-DomeCortefCortenemaCorticotropinCortisolCortisprayCortrilCuesDataDermacortDevelopmentEcological momentary assessmentEldecortEpidemiologyEtOH drinkingEtOH useExposure toFunctional MRIFunctional Magnetic Resonance ImagingFutureGoalsGustationHPA axisHPGFHepatocyte-Stimulating FactorHybridoma Growth FactorHydrocortisoneHydrocortoneHydrogen OxideHytoneIFN-beta 2IFNB2IL-1 betaIL-1 βIL-1-bIL-1βIL-6IL1-BetaIL1-βIL1B ProteinIL1F2IL1βIL6 ProteinImmuneImmune responseImmunesImmunologic FactorsImmunological FactorsIndividualInflammatoryInflammatory ResponseInterleukin 1betaInterleukin-1 betaInterleukin-1βInterleukin-6InterventionInterviewLaboratoriesLifeMGI-2Macrophage-Derived TNFMeasuresMedialMonocyte-Derived TNFMotivationMyeloid Differentiation-Inducing ProteinNeurobiologyNeuroendocrineNeuroendocrine SystemNeurosecretory SystemsNutracortOxidative StressParticipantPersonsPhysiologicPhysiologicalPlasmacytoma Growth FactorPrefrontal CortexPreinterleukin 1 BetaProceduresProcessProctocortRelapseReportingRiskRisk FactorsRisk MarkerRoleSample SizeScanningSeveritiesStimulusSurvey InstrumentSurveysTNFTNF ATNF AlphaTNF geneTNF-αTNFATNFαTasteTaste PerceptionTestingTimeTumor Necrosis FactorTumor Necrosis Factor-alphaVentral Tegmental AreaVisualVulnerable PopulationsWaterWorkactive followupadulthoodagesalcohol cuealcohol ingestionalcohol intakealcohol product usealcohol related cuealcohol responsealcohol usealcohol use disorderalcoholic beverage consumptionalcoholic drink intakebehavior changebinge alcohol consumptionbinge drinkerbinge drinkingbio-markersbiologic markerbiomarkerbout drinkercravingcue reactivitycytokinedevelopmentaldrinkingdrinking behaviorepidemiologicepidemiologicalepisodic drinkerepisodic drinkingethanol consumptionethanol cueethanol drinkingethanol ingestionethanol intakeethanol product useethanol responseethanol useethanol use disorderfMRIfMRI scanfollow upfollow-upfollowed upfollowupfunctional MRI scanfunctional magnetic resonance imaging scangustatory perceptiongustatory processinggustatory responsehost responsehypothalamic-pituitary-adrenal (HPA) axishypothalamic-pituitary-adrenal axishypothalmus-pituitary-adrenal axisimmune system responseimmunologic substanceimmunological substanceimmunoresponseinterferon beta 2multi-modalitymultimodalityneuralneurobiologicalprospectiveresponseresponse to alcoholresponse to ethanolrisk predictorrisk predictorssexsocialsocial rolesocio-demographic variablessociodemographic variablestaste processingtaste responsetime intervalventral tegmentumvulnerable groupvulnerable individualvulnerable people
Description preview
Abstract
Binge Drinking (BD) is a known risk factor for Alcohol Use Disorders (AUDs). Why some people
“mature out” of BD while others persist and develop AUDs may not only be related to the adoption of adult
social roles, but may also be due to one’s physiological response to BD. One mechanism contributing to BD
appears to be enhanced cue…
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